Lyme disease is on the rise. Caused by a spirochete Borreliella burgdorferi, it affects an estimated 500,000 people in the United States alone. The antibiotics currently used to treat Lyme disease are broad spectrum, damage the microbiome, and select for resistance in non-target bacteria. We therefore sought to identify a compound acting selectively against B. burgdorferi. A screen of soil micro-organisms revealed a compound highly selective against spirochetes, including B. burgdorferi. Unexpectedly, this compound was determined to be hygromycin A, a known antimicrobial produced by Streptomyces hygroscopicus. Hygromycin A targets the ribosomes and is taken up by B. burgdorferi, explaining its selectivity. Hygromycin A cleared the B. burgdorferi infection in mice, including animals that ingested the compound in a bait, and was less disruptive to the fecal microbiome than clinically relevant antibiotics. This selective antibiotic holds the promise of providing a better therapeutic for Lyme disease and eradicating it in the environment.

The reports of athletes who suddenly collapse have been increasing noticeably lately. Heart problems such as heart inflammation are often the cause – one of the known life-threatening side effects of Covid vaccines, which even the manufacturers themselves warn against. The current phenomenon is also evident if you simply look on Wikipedia at the list of footballers who have collapsed and died. The year 2021 stands out with 13 entries so far. In no other year mentioned have more footballers died during a game. And this list goes back to the year 1889. So it really is a historical event.

The mainstream media is curiously uninterested in this major global story. The German online outlet Wochenblick compiled a referenced list of the cardiac incidents in October while another online outlet Granite Grok published a new list of sportsmen collapsing on the field. Other outlets also listed these incidents, with some cases overlapping.

""There are 479,000 published variants of the imaginary genome because it doesn’t exist. Every time the computer does it, it makes a new genome. The California Health Department, in writing, said, “They have no analysis whatsoever of any Delta variant.” This is a spell. It’s a big old anti-scientific hoax."

Posted 19 November 2020 | By Kari Oakes 

 

We present here our research on the presence of graphene in covid vaccines. We have carried out a random screening of graphene-like nanoparticles visible at the optical microscopy in seven random samples of vials from four different trademarks, coupling images with their spectral signatures of RAMAN vibration. By this technique, called micro-RAMAN, we have been able to determine the presence of graphene in some of these samples, after screening more than 110 objects selected for their graphene-like appearance under optical microscopy.

Hyperinflammation plays an important role in severe and critical COVID-19. Using inconsistent criteria, many researchers definehyperinflammation as a form of very severe inflammation with cytokine storm. Therefore, COVID-19 patients are treated withanti-inflammatory drugs. These drugs appear to be less efficacious than expected and are sometimes accompanied by seriousadverse effects. SARS-CoV-2 promotes cellular ATP release. Increased levels of extracellular ATP activate the purinergicreceptors of the immune cells initiating the physiologic pro-inflammatory immune response. Persisting viral infection drivesthe ATP release even further leading to the activation of the P2X7 purinergic receptors (P2X7Rs) and a severe yet physiologicinflammation.

Disease progression promotes prolonged vigorous activation of the P2X7R causing cell death and uncontrolledATP release leading to cytokine storm and desensitisation of all other purinergic receptors of the immune cells. This results inimmune paralysis with co-infections or secondary infections. We refer to this pathologic condition as hyperinflammation. Thereadily available and affordable P2X7R antagonist lidocaine can abrogate hyperinflammation and restore the normal immunefunction. The issue is that the half-maximal effective concentration for P2X7R inhibition of lidocaine is much higher than themaximal tolerable plasma concentration where adverse effects start to develop. To overcome this, we selectively inhibit theP2X7Rs of the immune cells of the lymphatic system inducing clonal expansion of Tregs in local lymph nodes. Subsequently,these Tregs migrate throughout the body exerting anti-inflammatory activities suppressing systemic and (distant) localhyperinflammation. We illustrate this with six critically ill COVID-19 patients treated with lidocaine.

https://www.ukcolumn.org/video/independent-undertaker-john-olooney-exposes-the-covid-19-pandemic

“It is easier to ride on the back of a tiger than to climb off it.”

This Russian proverb, which appears at the beginning of Dr. Harold Hillman’s last ever book, sums up the current state of our scientific establishment. Whether medicine, physics, or archaeology, all facets of scientific research rest on certain dogmas.

In essence, what this proverb is saying, is that it’s easier to carry on perpetuating falsities than it is to admit you were wrong, drop everything and start again from scratch.

Dr Harold Hillman writes:–
The only real guarantee of the progress of knowledge is for academics to be ready to enter into unlimited dialogue about their research and theories, especially about those which they have published. An academic who is not prepared to discuss or correspond with other interested parties is behaving improperly, and such conduct should not be tolerated by the academic community. Some colleagues seem to think that if they ignore the awkward questions about their disciplines, or are hostile to those who ask them, the contradictions or anomalies in their work will somehow or other resolve themselves, and their research can progress. On the contrary, such an attitude inhibits the examination of the fundamental aspects of their disciplines, and thus delays substantial progress.
    The following questions have never been answered satisfactorily, several of them never at all:"

47 UNANSWERED QUESTIONS IN BIOLOGY. 27 May 1996. June/July 1996

https://drsambailey.com/wp-content/uploads/2021/11/COVID-19-FRAUD-WAR-ON-HUMANITY.pdf

"Bayer executive admits in public that mRNAs are gene therapies and that, without the pandemic, no one would have been willing to have them injected."

https://mobile.twitter.com/roys351/status/1457484236685029379

"Today, Jenners’ experiments are regarded as a gross violation of medical ethics."

"Although Jenner’s vaccine lacked any evidence of safety or efficacy, it was feverishly promoted to the masses. The Royal Jennerian Society for the Extirpation of Small-pox even prepared a notice to be given by clergymen at the baptism of a child. The notice read, “As you value the life of your infant and the safety of your neighborhood, you will immediately avail yourself of the advantages offered [in vaccination]”[16].

The rollout of Jenner’s vaccine was referred to by mainstream scholars as a “widespread experiment”, which it undoubtedly was."

Severe acute respiratory syndrome coronavirus 2 (SARS–CoV–2) has led to the coronavirus disease 2019 (COVID–19) pandemic, severely affecting public health and the global economy. Adaptive immunity plays a crucial role in fighting against SARS–CoV–2 infection and directly influences the clinical outcomes of patients. Clinical studies have indicated that patients with severe COVID–19 exhibit delayed and weak adaptive immune responses; however, the mechanism by which SARS–CoV–2 impedes adaptive immunity remains unclear. Here, by using an in vitro cell line, we report that the SARS–CoV–2 spike protein significantly inhibits DNA damage repair, which is required for effective V(D)J recombination in adaptive immunity. Mechanistically, we found that the spike protein localizes in the nucleus and inhibits DNA damage repair by impeding key DNA repair protein BRCA1 and 53BP1 recruitment to the damage site. Our findings reveal a potential molecular mechanism by which the spike protein might impede adaptive immunity and underscore the potential side effects of full-length spike-based vaccines.